ADDITIONAL INFORMATION:
·
Character
chosen by Mendel: The genes for seven characters chosen
by Mendel are present on four chromosomes (i) Chromosome 1- Flower colour and
seed colour (ii) Chromosome 4- Flower position, pod shape and plant height
(iii) Chromosome 5- Pod colour (iv) Chromosome 7- Seed shape.
·
Isoalleles:
Alleles producing similar phenotype but distinguishable amongst themselves
through change options.
·
Psudoalleles:
Genes lying side by side, producing related phenotypic effect and
distinguishable through a rare crossing over. eg. star (dominant) and asteroid
(recessive) traits in Drosophila.
·
Kolreuter
(1960): Discovered inheritance to be due to particulate
factors. Father of polygenic inheritance.
·
Galton
(1883): coined the term eugenics.
·
Correns:
Co-rediscoverer of principles of heredity, gave the name of laws to two of the
observations of Mendel, discovered incomplete dominance in 1903, and
cytoplasmic inheritance in 1909.
·
W.
Johannsen: Coined the term pure line
(1900), gene (1909), genotype and phenotype (1911).
·
Shull
(1914): coined the term heterosis.
·
Bateson:
Credited with coining the terms heterozygote and homozygote, F1,F2
allele and genetics.
·
Nilsson
Ehle (1908): Experimentally proved
polygenic/quantitative inheritance for the first time.
·
Actinobiology:
Study
of effects of radiations on living
organisms.
·
Father
of Actionobiology: Mullar
·
Blakeslee
(1937): Discovered the effect of colchicines on induction of
polyploidy.
·
Winkler
(1916): Discovered aneuploidy.
·
Daltonism:
Alternate same of red-green colour blindness alter the famous scientist who was
afflicted with it.
·
Protoanopia:
Red
blindness
·
Deuteronopia:
Green blindness
·
Ishihora
cards: Cards used for checking colour blindness.
·
Gynandromorphs:
Individual with both male and female traits.
·
Glucose
6Phosphate Dehydrogenase Deficiency (G-6PD deficiency):It
occurs as inborn error of metabolism in some persons. It is also called favism, because beans cause haemolysis
in the person suffering from G-6PD deficiency. Antimalarial drugs like primaquin
causes haemolysis in such persons. The haemolysis is due to production of H2O2,
which is not removed because of G-6PD deficiency and the result is lack of
NADPH2. Malarial parasite cannot complete schizogony in G-6PD
deficient patients due to premature death of RBCs.
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